My work in the Keck Lab focused on an essential process in bacteria called DNA replication restart which rescues abandoned replication forks by reloading the DNA replication machinery.
This project examined the effects of hyperactive RecA on Δ priB cells' fitness in E.coli using Transposon Sequencing and a Growth Competition Assay.
This paper discusses the hypothetical results and implications of the expected results from the investigation of the role of NagC in double-stranded DNA break repair in E.coli.
This project proposal aimed to gain support for the investigation of the role of NagC in double-stranded DNA break repair in E.coli using RNA-sequencing and CHIP-sequencing.
My research at the Medical College of Wisconsin focused on wound healing in the vascular epithelium in diabetic rats. This project examined the effects of impaired cell signaling on cell proliferation and wound closure in diabetic patients.
This poster presented my findings on wound closure rates in the vascular epithelium of diabetic rats using a wound rake model to stimulate cell injury, which then allowed me to track cell proliferation rates to examine healing.
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